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Whilst exact mechanisms remain unclear, a combination of risk factor reduction in improved HbA1c, weight, metabolic profile, as well as direct effects of GLP-1 agonism on coronary endothelial function and improved microvascular perfusion is likely at play

Spine, 8(2), 131140

Gut 55(10):14151422 Carter D, Warsen A, Mandell K, Cuschieri J, Maier RV, Arbabi S (2014) Delayed topical p38 MAPK inhibition attenuates full-thickness burn wound inflammatory signaling

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Regulation of mouse sterol regulatory element-binding protein-1c gene (SREBP-1c) by oxysterol receptors, LXRalpha and LXRbeta